[1] |
Plutzer J, Karanis P. Genetic polymorphism in Cryptosporidium species: an update[J]. Vet Parasitol, 2009, 165(3/4): 187-199.
doi: 10.1016/j.vetpar.2009.07.003
|
[2] |
Striepen B. Parasitic infections: time to tackle cryptosporidiosis[J]. Nature, 2013, 503(7475): 189-191.
doi: 10.1038/503189a
|
[3] |
O’Donoghue PJ. Cryptosporidium and cryptosporidiosis in man and animals[J]. Int J Parasitol, 1995, 25(2): 139-195.
doi: 10.1016/0020-7519(94)E0059-V
|
[4] |
Farthing MJ. Clinical aspects of human cryptosporidiosis[J]. Contrib Microbiol, 2000, 6: 50-74.
pmid: 10943507
|
[5] |
Chen XM, Keithly JS, Paya CV, et al. Cryptosporidiosis[J]. N Engl J Med, 2002, 346(22): 1723-1731.
doi: 10.1056/NEJMra013170
|
[6] |
GBD diarrhoeal diseases collaborators. Estimates of global, regional, and national morbidity, mortality, and aetiologies of diarrhoeal diseases: a systematic analysis for the global burden of disease study 2015[J]. Lancet Infect Dis, 2017, 17(9): 909-948.
doi: 10.1016/S1473-3099(17)30276-1
|
[7] |
Shaw MK. Cell invasion by Theileria sporozoites[J]. Trends Parasitol, 2003, 19(1): 2-6.
doi: 10.1016/S1471-4922(02)00015-6
|
[8] |
O’Hara SP, Huang BQ, Chen XM, et al. Distribution of Cryptosporidium parvum sporozoite apical organelles during attachment to and internalization by cultured biliary epithelial cells[J]. J Parasitol, 2005, 91(5): 995-999.
doi: 10.1645/GE-495R.1
|
[9] |
Kaiko GE, Stappenbeck TS. Host-microbe interactions shaping the gastrointestinal environment[J]. Trends Immunol, 2014, 35(11): 538-548.
doi: 10.1016/j.it.2014.08.002
|
[10] |
Peterson LW, Artis D. Intestinal epithelial cells: regulators of barrier function and immune homeostasis[J]. Nat Rev Immunol, 2014, 14(3): 141-153.
doi: 10.1038/nri3608
pmid: 24566914
|
[11] |
Okumura R, Takeda K. Roles of intestinal epithelial cells in the maintenance of gut homeostasis[J]. Exp Mol Med, 2017, 49(5): e338.
doi: 10.1038/emm.2017.20
|
[12] |
Muñoz M, Eidenschenk C, Ota N, et al. Interleukin-22 induces interleukin-18 expression from epithelial cells during intestinal infection[J]. Immunity, 2015, 42(2): 321-331.
doi: 10.1016/j.immuni.2015.01.011
|
[13] |
Garcia-Carbonell R, Yao SJ, Das S, et al. Dysregulation of intestinal epithelial cell RIPK pathways promotes chronic inflammation in the IBD gut[J]. Front Immunol, 2019, 10: 1094.
doi: 10.3389/fimmu.2019.01094
pmid: 31164887
|
[14] |
Lantier L, Lacroix-Lamandé S, Potiron L, et al. Intestinal CD103+ dendritic cells are key players in the innate immune control of Cryptosporidium parvum infection in neonatal mice[J]. PLoS Pathog, 2013, 9(12): e1003801.
doi: 10.1371/journal.ppat.1003801
|
[15] |
Ming ZP, Wang Y, Gong AY, et al. Attenuation of intestinal epithelial cell migration during Cryptosporidium parvum infection involves parasite Cdg7_FLc_1030 RNA-mediated induction and release of dickkopf-1[J]. J Infect Dis, 2018, 218(8): 1336-1347.
doi: 10.1093/infdis/jiy299
|
[16] |
Ming ZP, Gong AY, Wang Y, et al. Trans-suppression of host CDH3 and LOXL4 genes during Cryptosporidium parvum infection involves nuclear delivery of parasite Cdg7_FLc_1000 RNA[J]. Int J Parasitol, 2018, 48(6): 423-431.
doi: 10.1016/j.ijpara.2017.10.008
|
[17] |
Clemson CM, Hutchinson JN, Sara SA, et al. An architectural role for a nuclear noncoding RNA: NEAT1 RNA is essential for the structure of paraspeckles[J]. Mol Cell, 2009, 33(6): 717-726.
doi: 10.1016/j.molcel.2009.01.026
|
[18] |
Yamazaki T, Souquere S, Chujo T, et al. Functional domains of NEAT1 architectural lncRNA induce paraspeckle assembly through phase separation[J]. Mol Cell. 2018; 70(6): 1038-1053.e7.
doi: 10.1016/j.molcel.2018.05.019
|
[19] |
Nakagawa S, Naganuma T, Shioi G, et al. Paraspeckles are subpopulation-specific nuclear bodies that are not essential in mice[J]. J Cell Biol, 2011, 193(1): 31-39.
doi: 10.1083/jcb.201011110
|
[20] |
Fox AH, Nakagawa S, Hirose T, et al. Paraspeckles: where long noncoding RNA meets phase separation[J]. Trends Biochem Sci 2018, 43(2): 124-135.
doi: 10.1016/j.tibs.2017.12.001
|
[21] |
Wang L, Qu P, Yin WL, et al. Lnc-NEAT1 induces cell apoptosis and inflammation but inhibits proliferation in a cellular model of hepatic ischemia/reperfusion injury[J]. J Int Med Res, 2021, 49(3): 300060519887251.
|
[22] |
Liu ZH, Lu T, Liu SM, et al. Long non-coding RNA NEAT1 contributes to lipopolysaccharide-induced inflammation and apoptosis of human middle ear epithelial cells via regulating the miR-301b-3p/TLR4 axis[J]. Exp Ther Med, 2021, 22(6): 1360.
doi: 10.3892/etm.2021.10795
|
[23] |
Wang ZQ, Li K, Huang WR. Long non-coding RNA NEAT1-centric gene regulation[J]. Cell Mol Life Sci, 2020, 77(19): 3769-3779.
doi: 10.1007/s00018-020-03503-0
|
[24] |
Chen Y, Qiu JL, Chen B, et al. RETRACTED: long non-coding RNA NEAT1 plays an important role in Sepsis-induced acute kidney injury by targeting miR-204 and modulating the NF-κB pathway[J]. Int Immunopharm, 2018, 59: 252-260.
doi: 10.1016/j.intimp.2018.03.023
|
[25] |
Zhang P, Cao L, Zhou R, et al. The lncRNA NEAT1 promotes activation of inflammasomes in macrophages[J]? Nat Commun, 2019, 10(1): 1495.
|
[26] |
Zhang FF, Shen N, Tang YJ. Identification LncRNA NEAT1 as a new mediator of the TLR2-induced cytokine production[J]. Current Immunol, 2015, 35(4): 316-321. (in Chinese)
|
|
( 张飞飞, 沈南, 唐元家. LncRNA NEAT1参与TLR2介导的炎症因子的表达[J]. 现代免疫学, 2015, 35(4): 316-321.)
|
[27] |
Li J, Moran T, Swanson E, et al. Regulation of IL-8 and IL-1beta expression in Crohn’s disease associated NOD2/CARD15 mutations[J]. Hum Mol Genet, 2004, 13(16): 1715-1725.
doi: 10.1093/hmg/ddh182
|
[28] |
Matson JP, Dumitru R, Coryell P, et al. Rapid DNA replication origin licensing protects stem cell pluripotency[J]. eLife, 2017, 6: e30473.
doi: 10.7554/eLife.30473
|
[29] |
Zeng CW, Liu SC, Lu S, et al. The c-myc-regulated lncRNA NEAT1 and paraspeckles modulate imatinib-induced apoptosis in CML cells[J]. Mol Cancer, 2018, 17(1): 130.
doi: 10.1186/s12943-018-0884-z
|
[30] |
Yu X, Li Z, Zheng HY, et al. NEAT1: a novel cancer-related long non-coding RNA[J]. Cell Prolif, 2017, 50(2): e12329.
doi: 10.1111/cpr.12329
|
[31] |
Li WJ, Zhang ZH, Liu XH, et al. The FOXN3-NEAT1-SIN3A repressor complex promotes progression of hormonally responsive breast cancer[J]. J Clin Invest, 2017, 127(9): 3421-3440.
doi: 10.1172/JCI94233
|
[32] |
Zhang J, Guo S, Piao HY, et al. ALKBH5 promotes invasion and metastasis of gastric cancer by decreasing methylation of the lncRNA NEAT1[J]. J Physiol Biochem, 2019, 75(3): 379-389.
doi: 10.1007/s13105-019-00690-8
pmid: 31290116
|
[33] |
Zeng CW, Xu Y, Xu L, et al. Inhibition of long non-coding RNA NEAT1 impairs myeloid differentiation in acute promyelocytic leukemia cells[J]. BMC Cancer, 2014, 14: 693.
doi: 10.1186/1471-2407-14-693
|
[34] |
Imamura K, Imamachi N, Akizuki G, et al. Long noncoding RNA NEAT1-dependent SFPQ relocation from promoter region to paraspeckle mediates IL-8 expression upon immune stimuli[J]. Mol Cell, 2014, 53(3): 393-406.
doi: 10.1016/j.molcel.2014.01.009
|